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Contemporary OMFS (2008, p. 337)
Normal Pulp: Asymptomatic; responds normally to vitality tests.
Reversible Pulpitis: Pain to stimuli (like cold) that resolves immediately upon removal.
Symptomatic Irreversible Pulpitis: Vital pulp that cannot heal; characterized by lingering pain to thermal stimuli, spontaneous pain, or referred pain.
Asymptomatic Irreversible Pulpitis: Vital pulp that cannot heal, but currently has no clinical symptoms (usually due to deep caries or trauma).
Pulp Necrosis: The pulp is dead; non-responsive to vitality testing.
Previously Treated: The tooth has had a prior root canal or endodontic therapy.
Previously Initiated Therapy: A partial endodontic treatment has been started (e.g., pulpotomy or pulpectomy).
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Normal Apical Tissues: Not sensitive to percussion or palpation; lamina dura is intact.
Symptomatic Apical Periodontitis: Painful response to biting or percussion. May or may not have a radiographic apical lucency.
Asymptomatic Apical Periodontitis: Inflammation and destruction of apical periodontium (radiolucency present) but no clinical symptoms.
Chronic Apical Abscess: Inflammatory reaction to pulpal infection; characterized by gradual onset and a sinus tract (stoma). Usually painless.
Acute Apical Abscess: Inflammatory reaction to pulpal necrosis; characterized by rapid onset, spontaneous pain, swelling, and often systemic fever or malaise.
Condensing Osteitis: Diffuse radiopaque lesion representing a localized bony reaction to a low-grade inflammatory stimulus.
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Non-Restorable Caries: Decay has progressed to a point where the tooth cannot be functionally restored (e.g., subgingival or into the furcation).
Vertical Root Fracture (VRF): A fracture beginning on the root surface; usually necessitates extraction.
Cracked Tooth Syndrome: An incomplete fracture of a vital posterior tooth that may or may not involve the pulp.
Severe Periodontitis: Class III mobility or bone loss exceeding 70% with furcation involvement.
Internal/External Resorption: Pathological loss of tooth structure originating from the pulp or PDL.
Impacted: (Specific to 3rd molars) Classified as Soft Tissue, Partial Bony, or Complete Bony Impaction.
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4. Prognosis: Action/Plan
Good│Stable Routine Maintenance
Fair│Monitor/Early Intervention S&RP / Local Delivery Antimicrobials
Poor/Guarded │High Risk Surgical Consult / Periodontal Surgery
Questionable│Low Predictability Consider Extraction vs. Heroic Dentistry
Hopeless│Non-Functional/Infection Risk Extraction & Grafting / Implant Site Prep
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Example » Diagnosis #19: Pulpal Necrosis with Symptomatic Apical Periodontitis; Non-restorable due to Vertical Root Fracture. Prognosis: Hopeless.
Common Terms:
Vitality
Vital, Non-Vital, Necrotic
Response
Lingering, Non-Lingering, Spontaneous
Apical
Percussion (+/-), Palpation (+/-), Swelling (+/-)
Integrity
Fractured, Non-Restorable, Perforated
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Periodontal DDX │
American Academy of Periodontology »
Periodontal DDx (AAP Staging/Grading)
Instead of just "Gingivitis" vs "Periodontitis," use the Stage/Grade system to justify your surgical treatment plans (like implants or grafting).
Periodontal Health: Pink, firm, no bleeding, no attachment loss.
Gingivitis: Biofilm-induced inflammation without clinical attachment loss (CAL).
Periodontitis (The "Surgical" Tiers):
Stage I/II (Mild-Moderate): Horizontal bone loss, Probing Depths (PD) 5 mm. Usually manageable with ScRP.
Stage III (Severe): Vertical bone loss, PD 6 mm, furcation Grade II or III. surgical consult
Stage IV (Advanced/Potential Tooth Loss):Significant tooth loose, masticatory dysfunction, complex rehab,
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DDX Visual/Clinical Action
Plaque-Induced Gingivitis Bleeding, No Bone Loss Prophy / OHI
Stage III/IV Perio Deep pockets, Vertical defects Surgical Intervention
Endo-Perio Lesion Deep pocket + Non-vital pulp Endo first, then Re-eva
Vertical Root Fracture Isolated deep pocket, hx or Rct Extraction (Hopeless)
Nerotizing Periodontal Disease Punched out" papillae, extreme pain Antibiotics + Debridement
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Early furcation involvement
Slight bone loss in the furcation area
Pocket extends into furcation slightly
Minimal horizontal penetration
Partial horizontal bone loss
Furcation can be entered with an explorer/probe
Cul-de-sac involvement
Does not extend completely through the tooth
Complete horizontal furcation involvement
Probe can pass entirely through the furcation
Furcation often covered by soft tissue clinically
Same osseous destruction as Grade III
Gingival recession exposes the furcation clinically
Furcation is visibly open and accessible
Endo│ Chairside Reference »